Mun kortisolit alkaa nyt olee ihan huipussaan, enkä enää jaksa puhtaakskirjottaa noita ajatusvyöryjä. Ei mikään ihme jos ei kaikki mun pointit ihan mee perille.
Kuitenkin.
-On mahdollista (ainakin jossain määrin) kontrolloida urheilun kannalta tärkeitä arvoja kehossa. Tarkemmin sanottuna, niiden piikkejä. Ihan turha luulla, että saa psynteesin toimimaaan koko päivän, mutta sen saa toimimaan johonkin aikaan tosi voimakkaasti. Tai että testot olis aamusta iltaan maximeissa.
-Kolmella terialla se ei onnistu niin hyvin kuin kuudella.
-Kolmesta ateriasta ei saa mitään etua verrattuna kuuteen.
"These data extend growing evidence of a link between stress response systems and human eating behavior, by suggesting that activity within the HPA axis – our central, neuroendocrine stress response system – is neurobiologically linked to food consumption."
"No data on cortisol levels at the time of eating was collected, and the literature is contradictory as to whether the types of hassles documented are likely to elicit sufficient cortisol release to drive eating behavior (Dickerson and Kemeny 2004; van Eck et al., 1996). Therefore, in both cases, the mechanisms linking stress reactivity to eating behavior remained unclear."
"Whether the relationship between cortisol release and food intake in the current study involved direct physiological or pharmacological effects of GCs or the stimulation of other intermediary mechanisms is not clear at this point. However, the results do provide strong support for a contributing role of glucocorticoid activation in promoting food intake and perhaps thus in stimulating stress-related eating."
"It is also possible that the cortisol response to CRH provides a marker for some other factor that influences eating behavior. Epel et al (2001) acknowledge that in their study cortisol may have directly stimulated food intake, but the cortisol response to the laboratory stressor may also be marking some other trait vulnerability that also affects appetite."
"These data thus represent strong evidence that endogenously released cortisol might directly stimulate food consumption in humans."
"Both animal and other human data support a direct role for glucocorticoids in appetite regulation. "
"Increased glucocorticoid levels are associated with increased insulin secretion (Strack et al., 1995). The combined effects of glucocorticoids on consumption of high energy density foods and increased likelihood that energy consumed will be stored as fat in the presence of insulin means that HPA hyperactivity may be a mechanism by which obesity both occurs and is subsequently maintained. A recent study demonstrated that people who eat in response to stress show both elevated nocturnal insulin and cortisol (Epel et al., 2004) as well as high levels of weight gain. ‘Stress-eaters’ might, therefore, be at particularly great risk for developing and maintaining obesity and associated health problems."
"The precise mechanism linking food intake to cortisol reactivity in both psychosocial (Epel et al., 2001) and CRH challenge paradigms remains to be elucidated, but the current data support the hypothesis that cortisol itself may play a causal role in increasing food consumption. Further study is clearly needed to fully understand underlying mechanisms, but such work may prove useful in efforts to attack the severe public health crises created by obesity."
PubMed Central, Figure 2: Psychoneuroendocrinology. May 2010; 35(4): 607?612. Published online Oct 13, 2009. doi: 10.1016/j.psyneuen.2009.09.017
CRH-stimulated cortisol release and food intake in healthy, non-obese adults
